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Impaired myofilament contraction drives right ventricular failure secondary to pressure overload: model simulations, experimental validation, and treatment predictions

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Cardiovascular function and structure are preserved despite induced ablation of BMP1-related proteinases

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Organ-level right ventricular dysfunction with preserved Frank-Starling mechanism in a mouse model of pulmonary arterial hypertension

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Estrogen maintains mitochondrial content and function in the right ventricle of rats with pulmonary hypertension.

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Reduced haemodynamic coupling and exercise are associated with vascular stiffening in pulmonary arterial hypertension.

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Estrogen preserves pulsatile pulmonary arterial hemodynamics in pulmonary arterial hypertension.