Multi-scale Structure-Function Relationships in Right Ventricular Failure Due to Pressure Overload
/in Fibrosis, Hemodynamics, Pre-Clinical, RV Function, StressEarly Pulmonary Vascular Disease in Young Adults Born Preterm
/in Clinical, Hemodynamics, RV Function, Sex & Maturation, StressImpaired myofilament contraction drives right ventricular failure secondary to pressure overload: model simulations, experimental validation, and treatment predictions
/in Arterial Mechanics, Fibrosis, Hemodynamics, In Silico, Pre-Clinical, RV Function, StressCharacteristic Impedance: Frequency or Time Domain Approach.
/in Arterial Mechanics, Hemodynamics, In Silico, StressPulmonary vascular collagen content, not cross-linking, contributes to right ventricular pulsatile afterload and overload in early pulmonary hypertension.
/in Arterial Mechanics, Fibrosis, Genetics, Pre-Clinical, RV Function, StressReduced haemodynamic coupling and exercise are associated with vascular stiffening in pulmonary arterial hypertension.
/in Arterial Mechanics, Clinical, Hemodynamics, RV Function, StressEstrogen preserves pulsatile pulmonary arterial hemodynamics in pulmonary arterial hypertension.
/in Arterial Mechanics, Hemodynamics, Pre-Clinical, RV Function, Sex & Maturation, Stress17?-estradiol mediates superior adaptation of right ventricular function to acute strenuous exercise in female rats with severe pulmonary hypertension.
/in Arterial Mechanics, Fibrosis, Hemodynamics, Pre-Clinical, RV Function, Sex & Maturation, StressLimiting collagen turnover via collagenase-resistance attenuates right ventricular dysfunction and fibrosis in pulmonary arterial hypertension.
/in Fibrosis, Genetics, Hemodynamics, Pre-Clinical, RV Function, StressChesler Lab
Professor Naomi C. Chesler
University of California, Irvine
2420 Engineering Hall
(949) 824-3941
nchesler@uci.edu